Home >> Exams >> MD MS EE >> AIIMS MD MS EE >> AIIMS MD MS EE 2007 May >> ForumTopic: hiiiiiiii
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| Author: | Message |
| eagle Total Posts: 15 | Posted: Sat Aug 04, 2007 11:25 pm hiii, answer should be angioneurotic edema instead of foreign body. |
| eagle Total Posts: 15 | Posted: Sat Aug 11, 2007 03:11 am HELLOW FRIENDS, ]differentiation between acquired and hereditary angioneurotic edema is done on basis of- 1.C1q 2.C2 3.C4 4.C1-INH what do u think? |
| khuki Total Posts: 18 | Posted: Sat Aug 11, 2007 01:40 pm differentiation is done on the basis of c1-inh protein.hereditary angioneurotic edema is autosomal dominant and is of thrree types.in type 1 protein is deficient , in type 2 protein is dysfunctional and in type 3 pretein levels are normal and functional but still displays signs and sypmtoms of HAE. |
| eagle Total Posts: 15 | Posted: Sun Aug 12, 2007 04:47 am hellow friend, good try.c1 esterase inhibitor is decreased in hereditary angioneurotic edema.it is also decreased in acquired angioneurotic edema.then what is the answer?so confused. type 1 HAE present in 70-80percent cases in with c1INH IS decreased.c2 and c4 levels are particularely decreased in both during acut attacks. so differntiation is done on basis of c1q level.it is low iN AAE. IN HAE C1Q LEVEL MAY BE DECREASED BUT IT IS GREATER THAN 50PERCENT OF NORMAL.WHILE IN CASE OF AAE IT IS USUALLY LESS THAN 10PERCENT OF REFERENCE VALUE. |